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vrijdag 28 september 2018

Nature Reviews Neurology - Table of Contents alert Volume 14 Issue 10

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Nature Reviews Neurology
TABLE OF CONTENTS

October 2018 Volume 14, Issue 10

Research Highlights
News & Views
Reviews
 
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Research Highlights

 
Does tau pathology activate jumping genes?
Charlotte Ridler

p569 | doi:10.1038/s41582-018-0056-1
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FDA approves patisiran to treat hereditary transthyretin amyloidosis
Heather Wood

p570 | doi:10.1038/s41582-018-0065-0
PDF


 
Addition of bevacizumab fails to improve on temozolomide alone for glioma therapy
Heather Wood

p570 | doi:10.1038/s41582-018-0067-y
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A potential biomarker strategy to monitor treatment response in spinal muscular atrophy
Heather Wood

p570 | doi:10.1038/s41582-018-0068-x
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Traumatic brain injury induces transmissible tau pathology
Heather Wood

pp570 - 571 | doi:10.1038/s41582-018-0062-3
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APOE*ε4 — a risk factor for TDP43 pathology?
Ian Fyfe

pp570 - 571 | doi:10.1038/s41582-018-0064-1
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A new subtype of multiple sclerosis?
Ian Fyfe

p571 | doi:10.1038/s41582-018-0069-9
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MicroRNA from dying neurons triggers astrocytosis in ALS
Charlotte Ridler

p572 | doi:10.1038/s41582-018-0052-5
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Artificial intelligence accelerates detection of neurological illness
Charlotte Ridler

p572 | doi:10.1038/s41582-018-0066-z
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News & Views

 
Patent foramen ovale increases stroke risk in older people
Heinrich P. Mattle & Jeffrey L. Saver

pp573 - 574 | doi:10.1038/s41582-018-0050-7
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Multimodal imaging in RBD — present and future
Bradley F. Boeve & Kejal Kantarci

pp574 - 576 | doi:10.1038/s41582-018-0054-3
PDF


 
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Reviews

 
Neurofilaments as biomarkers in neurological disorders   
Michael Khalil, Charlotte E. Teunissen, Markus Otto, Fredrik Piehl, Maria Pia Sormani et al.

pp577 - 589 | doi:10.1038/s41582-018-0058-z
In this Review, Khalil et al. consider how technological advances have enabled the detection of neurofilament proteins in the blood, and discuss how these proteins consequently have the potential to be easily measured biomarkers of neuroaxonal injury in various neurological conditions.
Full Text | PDF


 
Spinocerebellar ataxias: prospects and challenges for therapy development   
Tetsuo Ashizawa, Gülin Öz & Henry L. Paulson

pp590 - 605 | doi:10.1038/s41582-018-0051-6
Advances in our understanding of the pathogenic mechanisms of spinocerebellar ataxias (SCAs) have resulted in the development of promising therapeutic strategies for these inherited neurodegenerative disorders. Here, Ashizawa and colleagues review the current progress and future challenges facing clinical trials of new therapies for the SCAs.
Full Text | PDF


 
Lafora disease — from pathogenesis to treatment strategies   
Felix Nitschke, Saija J. Ahonen, Silvia Nitschke, Sharmistha Mitra & Berge A. Minassian

pp606 - 617 | doi:10.1038/s41582-018-0057-0
Lafora disease is an autosomal recessive, progressive myoclonus epilepsy caused by loss of function of laforin or malin, leading to impaired glycogen metabolism. The authors review the clinical and molecular features of Lafora disease and discuss current and emerging treatment options.
Full Text | PDF
Collection: Rare diseases

 
Seizure prediction — ready for a new era   
Levin Kuhlmann, Klaus Lehnertz, Mark P. Richardson, Björn Schelter & Hitten P. Zaveri

pp618 - 630 | doi:10.1038/s41582-018-0055-2
In this Review, the authors consider advances over the past decade that have set the stage for a resurgence in attempts to predict seizures in epilepsy, and they propose new avenues of investigation that combine mechanisms, models, data, devices and algorithms.
Full Text | PDF


 
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